Search Results
70 items found for "pediatric tumors"
- Removing the GPCR-mediated brake on exocytosis enhances insulin action, promotes adipocyte browning, and protects against diet-induced obesity
Registration Logo Contest Committee Sponsors GPCR Retreat Program < Back to schedule Removing the GPCR-mediated Protease Activated Receptor signaling in the cardiovascular system and regulation of vesicular exocytosis mediated
- Coronavirus Porcine Epidemic Diarrhea Virus Utilizes Chemokine Interleukin-8 to Facilitate Viral Replication by Regulating Ca2+ Flux
chemokine interleukin-8 (CXCL8/IL-8) is indispensable for the activation and trafficking of inflammatory mediators and tumor progression and metastasis.
- Exploration of prognostic and treatment markers in hepatocellular carcinoma via GPCR-related genes analysis
It has been gradually recognized that GPCRs are of significance for tumorigenesis, but in-depth studies The tumor immune dysfunction and exclusion (TIDE) algorithm was utilized to evaluate immune infiltration
- Regulator of G protein signaling protein 6 alleviates acute lung injury by inhibiting inflammation and promoting cell self-renewal in mice
Regulator of G protein signaling protein 6 (RGS6), identified as a tumor suppressor gene, has received
- [Inhibitory effect of downregulating G protein-coupled receptor class C group 5 member A expression on lipopolysaccharide-induced inflammatory response in human gingival fibroblasts]
levels of the inflammatory cytokines in GFs induced by LPS, namely, interleukin (IL)-1β, IL-6, IL-8, tumor
- Ep 18 with Dr. Yamina Berchiche
Our guests were Maria Waldhoer , Tudor I. G protein-coupled receptors (GPCRs) mediate a vast variety of critical biological processes and provide
- Posters | Adhesion GPCR Workshop 2024 | Dr. GPCR Ecosystem
Generation and characterization of collecting duct specific GPR56 knockout mice Jianxiang Xue Anti-Tumorigenic Planar cell polarity (PCP) proteins contribute to tumour growth and invasion. differentially influence the stability of cell-cell and/or cell-matrix adhesions to dictate breast tumour carcinoma samples exhibiting non-invasive or invasive features, the latter including heterogeneous tumours Jianxiang Xue on the web Zaidman Physiology Lab Anti-Tumorigenic Role of Brain Angiogenesis Inhibitor
- Unveiling G-protein coupled receptors as potential targets for ovarian cancer nanomedicines: from RNA sequencing data analysis to in vitro validation
biopsies, matched with personalised GPCR-targeted nanomedicines, for the delivery of lethal drugs to tumour
- Full Agenda for Adhesion GPCR Workshop 2024 | Dr. GPCR Ecosystem
Van Meir Adhesion GPCR BAI1/ADGRB1 can block IGF1R-mediated growth signalling, increase radiosensitivity Of Complex Assembly And Ligand Interaction In The Adhesion GPCR Latrophilin/Cirl Demet Araç An ECR-Mediated Differentially Expressed Gpr116 (Adgrf5) Transcript Variants in Mouse Kidney Joseph Duman BAI1/ADGRB1-mediated Between Extracellular and Transmembrane Regions of a Holo-Adhesion GPCR Virginea de Araujo Farias Anti-Tumorigenic Medulloblastomas Alex Torrelli-Diljohn Investigating The Role of ADGRB3 Loss of Expression in Brain Tumor
- Receptor autoantibodies: Associations with cardiac markers, histology, and function in human non-ischaemic heart failure
Published date April 1, 2023 Abstract " Aims: A causal link between non-ischaemic heart failure (HF) and humoral
- Ep 154 with Dr Badr Sokrat
and Science Backgrounds Yamina and Badr discussed recording their podcast with Samary Eye, who made a humorous
- Investigating isoform switching in RHBDF2 and its role in neoplastic growth in breast cancer
the proteolysis mechanism is an irreversible process that aids in regulating cellular growth during tumorigenesis Speculatively, previous studies on gene expression analysis of RHBDF2 showed heterogenous behaviour during tumorigenesis Consistent with this, several studies have reported the antagonistic role of iRhom2 in tumorigenesis, Additionally, different opinions suggest iRhom2 mediated cleavage of EGFR ligands takes place TACE dependently
- GPR176 Promotes Cancer Progression by Interacting with G Protein GNAS to Restrain Cell Mitophagy in Colorectal Cancer
The GPR176/GNAS complex inhibits mitophagy via the cAMP/PKA/BNIP3L axis, thereby promoting the tumorigenesis
- RGS20 promotes non-small cell lung carcinoma proliferation via autophagy activation and inhibition of the PKA-Hippo signaling pathway
Lastly, in vivo experiments further confirmed role of RGS20 in aggravating tumorigenicity, as its overexpression