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In vitro, we used IFNα to stimulate human salivary gland epithelial cells (HSGECs), and the results showed that IFNα activated GRP78-ATF6-CHOP apoptosis signaling, decreased cell viability, and induced apoptosis

Stimulation of ectopically expressed muscarinic receptors induces IFN-γ but suppresses IL-2 production

GPCR Activation and Signaling Stimulation of ectopically expressed muscarinic receptors induces IFN-γ

stimulated/unstimulated HeLa S3 cells Since both TGFβ and IFNγ play an essential role in tumorigenesis importance to find out the regions undergoing histone modification changes in response to TGFβ and IFNγ Our study has compared the genes showing H3K4me3 occupancy in response to both TGFβ and IFNγ. Several genes were found to be shared between the TGFβ and IFNγ. DAVID Functional enrichment analysis in the TGFβ and IFNγ dataset revealed association of genes with

Specifically, the Mrgpra1-NPFF axis counter-regulated interferon (IFN) γ-mediated neutrophil polarization Distinct, cross-regulated populations of neutrophils were the primary source of NPFF and IFNγ during Yong , Aishwarya Magesh , Aishwarya Atakkatan , Felipe Andrade , Stephane Lajoie , Xinzhong Dong Tags IFNγ

Group 2 innate lymphoid cell , HES , High-affinity Fc receptor for IgE , Hypereosinophilic syndrome , IFNG-AS1 , IFNγ , IL , IL-13R , IL-13Rα1/2 , IL-1RαP , IL-31R , IL-31RA , IL-33R , IL-4Rα , IL-5Rα , ILC , ILC2