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25 items found for "Benign prostatic hyperplasia"
Posts (14)
- Exploiting Dependence of Castration-Resistant Prostate Cancer on the Arginine Vasopressin ...
September 2022 Exploiting Dependence of Castration-Resistant Prostate Cancer on the Arginine Vasopressin Signaling Axis by Repurposing Vaptans "Men with advanced prostate cancer are treated by androgen deprivation therapy but the disease recurs as incurable castration-resistant prostate cancer (CRPC), requiring new Interrogation of prostate cancer patient sample data revealed that coexpression of AVPR1A and AVPR2 is
- 📰 GPCR Weekly News, March 18 to 24, 2024
more Elucidation of active components and target mechanism in Jinqiancao granules for the treatment of prostatitis and benign prostatic hyperplasia GPCRs in Cardiology, Endocrinology, and Taste Blockade of endothelial
- Propranolol: A “Pick and Roll” Team Player in Benign Tumors and Cancer Therapies
), the therapeutic benefits of the β-adrenergic receptor antagonist, propranolol, were described in benign
Other Pages (11)
- Elucidation of active components and target mechanism in Jinqiancao granules for the treatment of prostatitis and benign prostatic hyperplasia
and benign prostatic hyperplasia Published date June 28, 2024 Abstract " Ethnopharmacological relevance : Prostatitis and benign prostatic hyperplasia (BPH) are inflammations of the prostate gland, which surrounds Aim of the study: We hypothesized that Jinqiancao granules could be a potential therapy for prostatitis Liangliang Zhou, Wenjie Yuan, Wanxian Wang, Yumin Yao, Jixia Wang, Yanfang Liu, Xinmiao Liang Tags Benign prostatic hyperplasia , G-protein coupled receptor , Jinqiancao granules , Network pharmacology , Prostatitis
- Autocrine proteinase-activated receptor signaling in PC3 prostate cancer cells
GPCR News < GPCRs in Oncology and Immunology Autocrine proteinase-activated receptor signaling in PC3 prostate
- In Silico Design of Novel RGS2-Galpha-q Interaction Inhibitors with Anticancer Activity
of the R4 family of RGS proteins, is overexpressed in many solid breast cancers, and its levels in prostate In addition, AJ-3 inhibited the migration of LNCaP prostate cancer cells in wound healing assays.