contributions to understanding the signaling, regulation and in vivo actions of the neuroprotective receptors GPR37 & GPR37L1 as well as the adhesion GPCRs BAI1, BAI2, and GPR56.

< GPCR News < GPCRs in Oncology and Immunology GPR97 depletion aggravates imiquimod-induced psoriasis In this research, our team allocated GPR97 depletion (GPR97-/-), GPR97 conditional depletion on dendritic cell (DC-cKO), and keratin 14-conditional knockout (K14-cKO) mice models to explore the function of GPR97 It was found that significantly aggravated psoriasis-like lesion in GPR97-/- mice. Particularly, we found that level of NF-κB p65 was increased in GPR97-/- DCs and BDP could inhibit p65

For instance, membrane receptor GPR97 is able to sense glucocorticoid to mediate its rapid actions, the

interact with a set of genomically related transmembrane receptors, like C5aR1 (CD88, C5aR) and C5aR2 (GPR77

CXCR4, GPR34, LGR6, LPAR3, and RGS2 were significantly expressed in three OV datasets and enabled accurate

Expression in Brain Tumor Formation in Li-Fraumeni Syndrome Tyler Bernadyn Elucidating The Role Of GPR97

Chang Cardiac Research Institute where he investigated the orphan G protein-coupled receptor (GPCR), GPR37L1