lymphoid cell , HES , High-affinity Fc receptor for IgE , Hypereosinophilic syndrome , IFNG-AS1 , IFNγ , IL , IL-13R , IL-13Rα1/2 , IL-1RαP , IL-31R , IL-31RA , IL-33R , IL-4Rα , IL-5Rα , ILC , ILC2 , IRF1 ,

Oncology and Immunology GPR97 depletion aggravates imiquimod-induced psoriasis pathogenesis via amplifying IL -23/IL-17 axis signal pathway Published date September 10, 2024 Abstract "Skin psoriasis is defined as receiving external stimulation to activate skin dendritic cells (DCs) which can release interleukin 23 (IL as induce T helper 17 (Th17) cell differentiation leading to elevated production of interleukin 17 (IL

and Immunology Stimulation of ectopically expressed muscarinic receptors induces IFN-γ but suppresses IL treatment induced high IFN-γ, CD69, and CD25 expression, but surprisingly did not induce substantial IL IL-2 3'UTR activity. This may explain the inhibitory impact on IL-2 production in hM3Dq/β1T cells. that activating the pAKT pathway is critical for IL-2 production in T cells."

Besides, when GPR109A was transiently silenced, the mRNA expression and secretion of IL-1β, IL-6 and

University, China 5 Department of Anatomy and Cell Biology, University of Illinois at Chicago, Chicago, IL

Department of Biochemistry and Molecular Biology, The University of Chicago, Chicago, IL, USA; 2. for Biophysical Dynamics, and Center for Mechanical Excitability, The University of Chicago, Chicago, IL Department of Molecular Biosciences, Northwestern University, Evanston, IL, USA; 4.