September 2022 GPR84 signaling promotes intestinal mucosal inflammation via enhancing NLRP3 inflammasome GPR84 activation imposes pro-inflammatory properties in colonic macrophages through enhancing NLRP3 inflammasome

disease therapy: unraveling the Foxg1-mediated neuroprotective mechanism through autophagy-mediated NLRP3

GPCR Activation and Signaling Butyrate potentiates Enterococcus faecalis lipoteichoic acid-induced inflammasome

The expression of an inflammasome marker NLRP3 upregulated by LPS was further elevated with prior exposure counteracting on the cAMP system, the common target for synaptamide and ethanol, and by regulating NLRP3 inflammasome." Tags Adenylyl cyclase , Cytokines , GPCR , Gavage , Lipopolysaccharide , Macrophages , Microglia , NLRP3 inflammasome , Phosphodiesterase , Synaptamide , cAMP.

., to adaptively initiate an apoptosis, inflammasome, Nrf2-ARE anti-oxidant, or heat-shock activation

HTR5A) and nucleotide-binding oligomerization domain-like receptor family pyrin domain containing 6 (NLRP6