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In this study, we evaluated the role of the astrocytic Gi pathway in neuroinflammation using a Gi -coupled Gi -DREADD was expressed in hippocampal astrocytes of a lipopolysaccharide (LPS)-induced neuroinflammation We found that astrocyte Gi -DREADD stimulation using clozapine N-oxide (CNO) inhibits neuroinflammation Taken together, our results indicate that the astrocytic Gi pathway plays an inhibitory role in neuroinflammation , providing an opportunity to identify potential cellular and molecular targets to control neuroinflammation

respond to cannabinoids which has implications for the development of cannabinoid-based treatments for neuroinflammation

Disorders GPCRs in Oncology and Immunology Exacerbating effects of single-dose acute ethanol exposure on neuroinflammation

perception, reproduction, development, growth, metabolism, and are also linked to major diseases, such as neuroinflammatory pro‐inflammatory disorders renders it a promising target in inflammatory and fibrotic conditions, including neuroinflammation

use disorders: Opportunities and hurdles for clinical development Chemokine CXCL13-CXCR5 signaling in neuroinflammation

Board of Directors Adiso Announces Groundbreaking Research Highlighting ADS024 a Novel Oral Agent for Neuroinflammation

Demonstration of the in vivo efficacy of a systemically administered antagonist analog in a model of neuroinflammation

promote morning wakefulness LPA3 agonist-producing Bacillus velezensis ADS024 is efficacious in multiple neuroinflammatory

multiple immunological cells such as microglia, macrophages and monocytes, and play a critical role in neuroinflammatory