Targeting these receptors preclinically can mitigate pathogenic β-amyloid and tau neurotoxicity whilst

Targeting these receptors preclinically can mitigate pathogenic β-amyloid and tau neurotoxicity whilst

carboxypeptidase E, CPE) and human 5-HTR1E, a G protein-coupled serotonin receptor with no previously known neurological diminished the NF-α1/CPE-mediated protection of these neurons against oxidative stress and glutamate neurotoxicity-induced

Odysseus Program Type I Program Grant to work as both the Adjunct Director of the VIB Center for Molecular Neurology ResearchGate Maudsley Lab on Facebook Receptor Biology Lab Facebook Group Twitter Semantic Scholar Instagram Neurotree

Odysseus Program Type I Program Grant to work as both the Adjunct Director of the VIB Center for Molecular Neurology ResearchGate Maudsley Lab on Facebook Receptor Biology Lab Facebook Group Twitter Semantic Scholar Instagram Neurotree

Odysseus Program Type I Program Grant to work as both the Adjunct Director of the VIB Center for Molecular Neurology ResearchGate Maudsley Lab on Facebook Receptor Biology Lab Facebook Group Twitter Semantic Scholar Instagram Neurotree