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acute activation of Gs signaling in skeletal muscle (SKM) in vivo and its contribution to whole-body glucose Results: Acute stimulation of GsD signaling in SKM impaired glucose tolerance in lean and obese mice by decreasing glucose uptake selectively into SKM. Clenbuterol injection improved glucose tolerance by increasing insulin secretion in lean mice. UCN2 injection resulted in decreased glucose tolerance associated with lower plasma insulin levels.

Objective: To study the effects of RH on metabolic pathways associated with glucose counterregulation Further, we performed pyruvate and lactate tolerance tests to assess gluconeogenesis. Finally, we conducted epinephrine tolerance test to investigate systemic glycemic excursions to counterregulatory

are thought to calibrate glucagon-like peptide 1 (GLP-1) bioavailability, thereby regulating systemic glucose the gut IEL GLP-1 receptor (GLP-1R) is not required for enteroendocrine L cell GLP-1 secretion and glucose Moreover, independent of glucose control or weight loss, the anti-inflammatory actions of GLP-1RAs require

diabetes mellitus (T2DM) is associated with low-grade chronic type 2 inflammation and disturbance of glucose Our results show that CB2 stimulation of ILC2s protects against insulin-resistance onset, ameliorates glucose tolerance, and reverses established insulin resistance. Hurrell, Omid Akbari Tags CB2 , CP: Immunology , CP: Metabolism , ILC2 , T2DM , adipose inflammation , glucose tolerance , immunotherapy , insulin resistance Source Contribute to the GPCR News Coming soon Become

< GPCR News < GPCRs in Oncology and Immunology Glucose and HODEs regulate Aspergillus ochraceus quorum sugar sensor, and intracellular adenylate cyclase (AcyA)-cAMP-PKA pathway that in response to ligands glucose

the role of immune cell GPCRs and their cross-talk with other insulin-sensitive tissues regulating glucose