diabetes mellitus (T2DM) is associated with low-grade chronic type 2 inflammation and disturbance of glucose Our results show that CB2 stimulation of ILC2s protects against insulin-resistance onset, ameliorates glucose tolerance, and reverses established insulin resistance. Hurrell, Omid Akbari Tags CB2 , CP: Immunology , CP: Metabolism , ILC2 , T2DM , adipose inflammation , glucose tolerance , immunotherapy , insulin resistance Source Contribute to the GPCR News Coming soon Become

< GPCR News < GPCRs in Oncology and Immunology Glucose and HODEs regulate Aspergillus ochraceus quorum sugar sensor, and intracellular adenylate cyclase (AcyA)-cAMP-PKA pathway that in response to ligands glucose

the role of immune cell GPCRs and their cross-talk with other insulin-sensitive tissues regulating glucose

Mutations in Sensitive Regions of Regulators of G Protein Signaling Predicted by Three-Dimensional Missense Tolerance RGS14 exhibited seven significantly tolerant and seven significantly intolerant residues, RGS10 had six intolerant residues, and RGS4 had eight tolerant and six intolerant residues. Intolerant and tolerant-control residues that overlap with pathogenic cancer mutations reported in the In downstream cAMP measurement, tolerant RGS14-D137Y and RGS10-S64T and intolerant RGS10-K89M resulted

No maximum tolerated dose was reached, and a regimen including mitoxantrone 18 mg/m2 per day and sorafenib

where she helped to investigate the role of various ATP-sensitive potassium channels in pain and opioid tolerance

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